Remember
that we have been going out in the midday sun for the whole of our existence
on this planet and like all the foods we have naturally eaten this form
of “nutrition” is part of our evolution and our metabolisms
have become dependant upon it, as we discussed last week.
So why has the incidence of skin cancers and malignant
melanomas increased in recent years?
As reported in The Nutrition Practitioner journal1
recently, “the medical practice of heliotherapy – exposing
patients to controlled amounts of sunlight to cure or alleviate various
illnesses – was accepted practice in many countries from the late
19th to mid-20th century”. Indeed, the 1903 Nobel Prize was given
to Dr Niels Finsen, a Danish Physician for his work using UV light to
treat tuberculosis (TB).
Several leading physicians followed suit with clinics popular in Switzerland,
the rest of Europe and on the south coast of England; you may have seen
the famous footage of the TB clinic on the Isle of Wight, built to maximise
exposure to the sun for thousands of patients. Dr Richard Hobday2
states on his website www.healingsun.com...
“Sunlight was prescribed by the physicians of Greece and Imperial
Rome. During the 19th century the health benefits of exposure began to
be investigated scientifically and during the first half of the 20th century
sunlight therapy was widely practised. Sunlight was used to disinfect
and heal the wounds of casualties during the First World War and in the
years that followed, heliotherapy (to use the medical term) was a popular
treatment for patients suffering from diseases such as tuberculosis and
rickets.”
It was only after the 1940s that these practices began to be maligned
and sun exposure linked to the rising incidence of skin cancers.
There is no dispute that this worrying statistic exists, but although
we are given the impression that UV exposure is completely to blame, why
the sudden rise after so many people found great benefit from heliotherapy?
We have to look at the dietary and environmental factors involved and
use these as a way to learn and protect ourselves against the downsides
of the cheapest health benefit we have. Simon Best writes that there has
been no evidence found that the decrease of the ozone layer at the poles
correlates with any increase in melanoma3. Citing
a study of Punta Arenas, the city closest to the Antarctic ozone hole
in South America, showed that the UV had “no noticeable effect”
on health problems4.
More importantly, research is now highlighting the link between
poor diets and an inability to counter UV damage; the early heliotherapists
advocated “gradual, daily exposure to the sun, coupled with a nutritious
diet, could cure many diseases and promote robust health”1.
Dr Rollier, who at one time had 36 clinics in Switzerland in the early
20th century, observed that skin that is lacking in minerals does not
respond as well as that which is fully nourished.
The rise of skin cancers has directly correlated with the decline
in the eating of fresh fruit and vegetables, beneficial oils and wholegrains.
This of course has also seen the shuffling in of processed foods
and higher levels of saturated and hydrogenated fats in foods. The hindering
of the correct healing and structure of the skin that this causes, predisposes
a person to sunburn and increases the risk of skin cancer.
It was reported in the New York Times5 this
month that a recent Australian study produced after ten years of observations,
found that there was “a 40 per cent reduction in melanoma among
sun worshippers who regularly ate fish”. Omega 3 oils found
in oily fish are potent anti-inflammatory agents and support the immune
system, so they may well be a factor in these statistics. It is important
for us to eat both omega 3 and omega 6 oils; termed “essential fats”
as we have to ingest them, cannot make them in the body and rely on them
for important body functions, correct metabolism and skin structure. These
are what waterproof us, keeping the skin supple, deficiencies often show
as dry, cracked or problem skin, easily illustrating how such skin would
be more prone to damage from intense UV light.
The ideal ratio for these fats is 2:1 of omega 3:6; omega 3 oils come
from mainly oily fish, but also some seeds, grains and beans, but here
they are also found with omega 6 oils. Our sources of these have increased
– they are found in plants or their oils – especially key
ingredients in processed foods; sunflower oil, safflower oil, vegetable
oil etc. This has resulted in some people eating a ratio of as much as
1:20 and most about 1:2; this especially applies to vegetarians who are
particularly advised to consider omega 3 supplementation.
Other nutritional factors for sun protection are:
| 1. | Dermatologist Roddie McKenzie at Edinburgh University suggests that the mineral selenium can protect the skin against UV damage. Its presence in the body as a protective antioxidant, helps to “quench” the oxidation that can lead to cancer. The National Diet and Nutrition Survey we discussed in a previous newsletter, shows that the average British diet lacks selenium – not surprising really, as non-organic soil has shown to be particularly depleted and it may not be getting into the usually good sources of brazil nuts, whole grains, sesame and sunflower seeds. Sources from fish may be better, but contamination of mercury in tuna may offset the benefits (to be discussed in a later newsletter). So, try to buy organic; again it has proven to be worth it. | ||||||||||||
| 2. | All antioxidants have been shown to be depleted in photoaged skin6. These are the vitamins A, C and E and the minerals zinc and selenium; these are found in fruit and vegetables, wholegrains, nuts, seeds, fish, chicken, red wine, green tea and pulses. At least achieving the minimum 5 portions a day of fruit and vegetables advised by the World Health Organisation is fundamental to health; this is conservative though, aim higher if you can and consider an antioxidant supplement. Variety is all important to achieve the full range and the following have particular significance: | ||||||||||||
| 3. | The carotenoids are a type of
antioxidant; they are colourful, oil-soluble pigments (colours) that
protect fatty tissues in the eyes, heart, skin, liver and kidneys
from damage. The most important forms for sun protection are6,7,8:
|
Total Being Opinion
Once again, it is back to as natural as possible. Why do we continue to
insist upon thinking we know better than Mother Nature? We were intended
to go out into the sun and have evolved with this exposure as an important
part of our metabolism. Our diets simultaneously allowed us to handle
the potentially very damaging UV light by containing the very powerful
protective agents in plants and fish. How do you think plants protect
themselves against UV when they rely on photosynthesis? How is it that
fish remain waterproofed and their skin has the same supple quality that
we equate with good skin health?
Make sure that you read the guidelines for safe exposure set out in Part
I of this article, go shopping for the foods mentioned above and then
think about running naked through the grass, full to the gills with delicious,
protective nutrients – feel healthier?
As a final note: do not use sun beds, they produce UVA and have been shown
to cause skin cancer, natural is always best9.
References
1. Best S. Why sunlight benefits outweigh hazards. Nut. Pract. Journal:
Part 1 Vol.4 Issue 2; Part 2 Vol.5 Issue 1
2. Hobday R. The Healing Sun. Findhorn Press, 1999
3. Br J Can. 1992; 65:916-21
4. Am J Pub Health 1995; 85(4): 546-50
5. Article: Melanoma: It’s down to omega-6, not the sun http://wddty.co.uk
6. Mukhtar H. Eat plenty of green leafy vegetables for photoprotection:
emerging evidence. J Invest Dermatol. 2003 Aug;121(2):399-405
7. Stahl W, Sies H. Carotenoids and protection against solar UV radiation.
Skin Pharmacol Appl Skin Physiol. 2002 Sep-Oct; 15(5):291-6. Review.
8. Lee J, Jiang S, Levine N, Watson R. Carotenoid supplementation reduces
erythema in human skin after simulated solar radiation exposure. Proc
Soc Exp Biolog Med. 2000 223:170-174.
9. Am J Epid. 1994; 140:691-9
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